Abstract The human placenta is critical throughout intrauterine development; it has a direct impact on the efficacy of fetal development, such as diabetes mellitus and obesity.1 Placental growth, differentiation and adaptation are influenced by maternal factors, such as stress, oxygen availability and maternal genetics. Placenta accreta is one of the catastrophic obstetric complications which leads to peripartum hysterectomy and common cause of severe maternal morbidity.1 This case report concerns a woman diagnosed with placenta previa with placenta accreta. She had been married for four years and had conceived three times, giving birth to two live babies and had one spontaneous abortion. She was presently admitted to the hospital with complaints of fever and chills from past one week with non-productive cough, vomiting from past one week with pain in abdomen and inability to pass urine. She had also history of IUD baby due to unmanageable labour pain at the time of delivery which was delivered by an unpractised and unlicensed nurse in the absence of obstetrician. Her last pregnancy was complicated by Placenta Previa and she had undergone for emergency caesarean section. Her chickenguniya test was found positive as she complained of fever with chills. Ultrasonography with NCCT correlation showed placenta previa with placenta percreta and uterine collection, calcified hematoma and calcified placental tissue along with bilateral enlarged kidneys with mild CMD attenuation. As a part of management, hysterectomy was performed. Keywords: Placenta Accreta; NCCT=Non- contrast computerized tomography; CMD = Corticomedullary differentiation.